Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Equipe Genetique des Anomalies du Developpement, |
RCV003224947 | SCV003920980 | likely pathogenic | Intellectual disability, autosomal dominant 14 | 2023-03-24 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV005102415 | SCV005769815 | pathogenic | not provided | 2024-12-24 | criteria provided, single submitter | clinical testing | This sequence change replaces tyrosine, which is neutral and polar, with cysteine, which is neutral and slightly polar, at codon 1096 of the ARID1A protein (p.Tyr1096Cys). This variant is not present in population databases (gnomAD no frequency). This missense change has been observed in individual(s) with clinical features of ARID1A-related conditions (internal data). In at least one individual the variant was observed to be de novo. ClinVar contains an entry for this variant (Variation ID: 2500289). Invitae Evidence Modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) indicates that this missense variant is expected to disrupt ARID1A protein function with a positive predictive value of 80%. For these reasons, this variant has been classified as Pathogenic. |