Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Mendelics | RCV000987423 | SCV001136713 | pathogenic | Retinitis pigmentosa 41 | 2019-05-28 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001858666 | SCV002239444 | pathogenic | not provided | 2023-12-11 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Ser290Ilefs*2) in the PROM1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in PROM1 are known to be pathogenic (PMID: 17605048, 19718270, 24154662, 25474345). This variant is present in population databases (no rsID available, gnomAD 0.003%). This premature translational stop signal has been observed in individuals with clinical features of autosomal recessive retinitis pigmentosa and/or cone rod dystrophy (PMID: 20042663, 31129250). It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 802055). For these reasons, this variant has been classified as Pathogenic. |
Ophthalmic Genetics Group, |
RCV003324542 | SCV004030274 | pathogenic | Cone-rod dystrophy | 2023-07-24 | criteria provided, single submitter | research | Clinical significance based on ACMG v2.0 |