Total submissions: 3
| Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
|---|---|---|---|---|---|---|---|---|
| Labcorp Genetics |
RCV001903543 | SCV002170127 | uncertain significance | not provided | 2022-07-12 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with leucine, which is neutral and non-polar, at codon 1204 of the PCNT protein (p.Pro1204Leu). This variant is present in population databases (rs749542536, gnomAD 0.008%). This variant has not been reported in the literature in individuals affected with PCNT-related conditions. ClinVar contains an entry for this variant (Variation ID: 1405063). Algorithms developed to predict the effect of missense changes on protein structure and function output the following: SIFT: "Tolerated"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0". The leucine amino acid residue is found in multiple mammalian species, which suggests that this missense change does not adversely affect protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
| Ambry Genetics | RCV002555389 | SCV003681452 | uncertain significance | Inborn genetic diseases | 2022-08-31 | criteria provided, single submitter | clinical testing | The c.3611C>T (p.P1204L) alteration is located in exon 19 (coding exon 19) of the PCNT gene. This alteration results from a C to T substitution at nucleotide position 3611, causing the proline (P) at amino acid position 1204 to be replaced by a leucine (L). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
| Prevention |
RCV004741136 | SCV005350035 | uncertain significance | PCNT-related disorder | 2024-03-28 | no assertion criteria provided | clinical testing | The PCNT c.3611C>T variant is predicted to result in the amino acid substitution p.Pro1204Leu. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0080% of alleles in individuals of African descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |