Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Genetic Services Laboratory, |
RCV000147151 | SCV000194512 | pathogenic | Microcephalic osteodysplastic primordial dwarfism type II | 2013-09-09 | criteria provided, single submitter | clinical testing | |
Baylor Genetics | RCV000147151 | SCV001523582 | pathogenic | Microcephalic osteodysplastic primordial dwarfism type II | 2019-09-16 | criteria provided, single submitter | clinical testing | This variant was determined to be pathogenic according to ACMG Guidelines, 2015 [PMID:25741868]. |
Labcorp Genetics |
RCV003556190 | SCV004298826 | pathogenic | not provided | 2023-11-27 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Glu1860*) in the PCNT gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in PCNT are known to be pathogenic (PMID: 18174396, 22821869). This variant is present in population databases (rs369195346, gnomAD 0.002%). This premature translational stop signal has been observed in individual(s) with microcephalic osteodysplastic primordial dwarfism type 2 (MOPD2) (PMID: 19643772). ClinVar contains an entry for this variant (Variation ID: 159621). For these reasons, this variant has been classified as Pathogenic. |