Total submissions: 7
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Quest Diagnostics Nichols Institute San Juan Capistrano | RCV000760029 | SCV000889779 | uncertain significance | not provided | 2018-04-07 | criteria provided, single submitter | clinical testing | |
Center for Genomics, |
RCV000768287 | SCV000898891 | uncertain significance | Facial dysmorphism-immunodeficiency-livedo-short stature syndrome | 2018-03-13 | criteria provided, single submitter | clinical testing | POLE NM_006231.3 exon 44 p.Thr2023Asn (c.6068C>A): This variant has not been reported in the literature but is present in 2/33552 Latino alleles in the Genome Aggregation Database (http://gnomad.broadinstitute.org/rs771628123). Evolutionary conservation and computational predictive tools for this variant are unclear. In summary, data on this variant is insufficient for disease classification. Therefore, the clinical significance of this variant is uncertain. |
Labcorp Genetics |
RCV000760029 | SCV000949132 | uncertain significance | not provided | 2024-10-20 | criteria provided, single submitter | clinical testing | This sequence change replaces threonine, which is neutral and polar, with asparagine, which is neutral and polar, at codon 2023 of the POLE protein (p.Thr2023Asn). This variant is present in population databases (rs771628123, gnomAD 0.006%). This variant has not been reported in the literature in individuals affected with POLE-related conditions. ClinVar contains an entry for this variant (Variation ID: 619906). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be disruptive. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Gene |
RCV000760029 | SCV001823319 | uncertain significance | not provided | 2023-09-06 | criteria provided, single submitter | clinical testing | Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis supports that this missense variant does not alter protein structure/function; Has not been previously published as pathogenic or benign to our knowledge; This variant is associated with the following publications: (PMID: 29056344) |
Ambry Genetics | RCV004027170 | SCV003874624 | uncertain significance | Hereditary cancer-predisposing syndrome | 2024-12-04 | criteria provided, single submitter | clinical testing | The p.T2023N variant (also known as c.6068C>A), located in coding exon 44 of the POLE gene, results from a C to A substitution at nucleotide position 6068. The threonine at codon 2023 is replaced by asparagine, an amino acid with similar properties. This amino acid position is highly conserved in available vertebrate species. In addition, this alteration is predicted to be tolerated by in silico analysis. Based on the available evidence, the clinical significance of this variant remains unclear. |
Center for Genomics, |
RCV003224386 | SCV003920342 | uncertain significance | Colorectal cancer, susceptibility to, 12; Facial dysmorphism-immunodeficiency-livedo-short stature syndrome; Intrauterine growth retardation, metaphyseal dysplasia, adrenal hypoplasia congenita, genital anomalies, and immunodeficiency | 2021-12-22 | criteria provided, single submitter | clinical testing | POLE NM_006231.3 exon 44 p.Thr2023Asn (c.6068C>A): This variant has not been reported in the literature but is present in 2/33552 Latino alleles in the Genome Aggregation Database (http://gnomad.broadinstitute.org/rs771628123). Evolutionary conservation and computational predictive tools for this variant are unclear. In summary, data on this variant is insufficient for disease classification. Therefore, the clinical significance of this variant is uncertain. |
Fulgent Genetics, |
RCV003224386 | SCV005633670 | uncertain significance | Colorectal cancer, susceptibility to, 12; Facial dysmorphism-immunodeficiency-livedo-short stature syndrome; Intrauterine growth retardation, metaphyseal dysplasia, adrenal hypoplasia congenita, genital anomalies, and immunodeficiency | 2024-05-04 | criteria provided, single submitter | clinical testing |