Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Revvity Omics, |
RCV001780209 | SCV002019884 | pathogenic | Retinitis pigmentosa 1 | 2020-09-02 | criteria provided, single submitter | clinical testing | |
Genomic Medicine Center of Excellence, |
RCV001780209 | SCV005375252 | likely pathogenic | Retinitis pigmentosa 1 | 2024-10-13 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (GRCh38; NM_006269.2:c.3428del:p.Asn1143IlefsTer25) in the RP1 protein. This alteration is expected to result in loss of function by premature termination codon resulting in protein truncation, or nonsense-mediated mRNA decay. This alteration is interpreted as disease-causing mutation, a commonly known mechanism for disease. Not observed at significant frequency in large population cohorts (gnomAD). ClinVar contains an entry for this variant (Variation ID: 979006). This variant is associated with the following publications: PubMed: 23105016, 22317909, 26355662 In summary, this variant meets our criteria for classification as Likely pathogenic based on the evidence outlined |
Faculty of Health Sciences, |
RCV001257890 | SCV001434658 | pathogenic | Autosomal recessive retinitis pigmentosa | 2015-09-10 | no assertion criteria provided | literature only |