Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000375478 | SCV000339319 | pathogenic | not provided | 2016-01-25 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000375478 | SCV002227852 | uncertain significance | not provided | 2022-08-23 | criteria provided, single submitter | clinical testing | This sequence change results in a frameshift in the UQCRB gene (p.Arg105Lysfs*22). While this is not anticipated to result in nonsense mediated decay, it is expected to disrupt the last 7 amino acid(s) of the UQCRB protein and extend the protein by 14 additional amino acid residues. This variant is present in population databases (no rsID available, gnomAD 0.01%). This frameshift has been observed in individual(s) with autosomal recessive mitochondrial complex III deficiency (PMID: 12709789, 28604960). ClinVar contains an entry for this variant (Variation ID: 286054). Algorithms developed to predict the effect of variants on protein structure and function are not available or were not evaluated for this variant. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Experimental studies have shown that this frameshift affects UQCRB function (PMID: 25446085). |
OMIM | RCV004567831 | SCV000033339 | pathogenic | Mitochondrial complex III deficiency nuclear type 3 | 2003-07-01 | no assertion criteria provided | literature only |