Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Blueprint Genetics | RCV001073414 | SCV001238955 | likely pathogenic | Retinal dystrophy | 2019-01-23 | criteria provided, single submitter | clinical testing | |
Ocular Genomics Institute, |
RCV001376396 | SCV001573519 | pathogenic | Retinitis pigmentosa 38 | 2021-04-08 | criteria provided, single submitter | research | The MERTK c.2049_2052del variant was identified in an individual with retinitis pigmentosa with a presumed recessive inheritance pattern. Through a review of available evidence we were able to apply the following criteria: PVS1, PM2, PM3. Based on this evidence we have classified this variant as Pathogenic. |
Labcorp Genetics |
RCV005056858 | SCV005693121 | pathogenic | not provided | 2024-03-12 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Leu683Phefs*18) in the MERTK gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in MERTK are known to be pathogenic (PMID: 24265693, 29659094). This variant is present in population databases (rs775776288, gnomAD 0.006%). This premature translational stop signal has been observed in individual(s) with inherited retinal disease (PMID: 31429209). ClinVar contains an entry for this variant (Variation ID: 865869). For these reasons, this variant has been classified as Pathogenic. |