Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Genetic Services Laboratory, |
RCV000147518 | SCV000194955 | likely pathogenic | Encephalopathy due to GLUT1 deficiency | 2013-06-06 | criteria provided, single submitter | clinical testing | |
3billion | RCV000147518 | SCV002012345 | pathogenic | Encephalopathy due to GLUT1 deficiency | 2021-10-02 | criteria provided, single submitter | clinical testing | Same nucleotide change resulting in same amino acid change has been previously reported as pathogenic/likely pathogenic with supporting evidence (ClinVar ID: VCV000159921.1, PS1). It is not observed in the gnomAD v2.1.1 dataset (PM2). A different missense change at the same codon (p.Asn34Ser) has been reported as pathogenic/likely pathogenic with strong evidence (ClinVar ID: VCV000662199.8, PMID: 23340081 and 28961260, PM5). The variant was observed as assumed (i.e. paternity and maternity not confirmed) de novoo (3billion dataset, PM6). Missense changes are a common disease-causing mechanism (PP2).In silico tool predictions suggest damaging effect of the variant on gene or gene product (REVEL: 0.743, 3Cnet: 0.848, PP3). Therefore, this variant is classified pathogenic according to the recommendation of ACMG/AMP guideline. |
Genome- |
RCV000147518 | SCV004173959 | likely pathogenic | Encephalopathy due to GLUT1 deficiency | 2023-04-11 | criteria provided, single submitter | clinical testing |