Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001704788 | SCV000729450 | likely benign | not provided | 2020-12-15 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000904143 | SCV001048646 | likely benign | GLUT1 deficiency syndrome 1, autosomal recessive | 2024-01-25 | criteria provided, single submitter | clinical testing | |
Center for Genomics, |
RCV003224350 | SCV003920482 | uncertain significance | Hereditary cryohydrocytosis with reduced stomatin; Dystonia 9; Encephalopathy due to GLUT1 deficiency; Childhood onset GLUT1 deficiency syndrome 2; Epilepsy, idiopathic generalized, susceptibility to, 12 | 2021-03-30 | criteria provided, single submitter | clinical testing | SLC2A1 NM_006516 exon 7 p.Ala301Ala (c.903G>A): This variant has not been reported in the literature but is present in 8/33504 Latino individuals in the Genome Aggregation Database (http://gnomad.broadinstitute.org/rs776461617). Evolutionary conservation and computational predictive tools for this variant are limited or unavailable. Of note, this variant is a silent variant and does not change the amino acid, reducing the probability that this variant is disease causing. In summary, data on this variant is insufficient for disease classification. Therefore, the clinical significance of this variant is uncertain. |
Prevention |
RCV004547752 | SCV004715766 | likely benign | SLC2A1-related disorder | 2020-12-28 | criteria provided, single submitter | clinical testing | This variant is classified as likely benign based on ACMG/AMP sequence variant interpretation guidelines (Richards et al. 2015 PMID: 25741868, with internal and published modifications). |