Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001048854 | SCV001212879 | uncertain significance | not provided | 2022-07-25 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with threonine, which is neutral and polar, at codon 68 of the KDM5B protein (p.Pro68Thr). This variant is present in population databases (rs369444528, gnomAD 0.01%). This missense change has been observed in individual(s) with clinical features of KDM5B-related conditions (Invitae). ClinVar contains an entry for this variant (Variation ID: 845733). Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Fulgent Genetics, |
RCV002479297 | SCV002792936 | uncertain significance | Intellectual disability, autosomal recessive 65 | 2021-07-31 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV004553588 | SCV004747166 | uncertain significance | KDM5B-related disorder | 2023-10-23 | no assertion criteria provided | clinical testing | The KDM5B c.202C>A variant is predicted to result in the amino acid substitution p.Pro68Thr. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0052% of alleles in individuals of East Asian descent in gnomAD (http://gnomad.broadinstitute.org/variant/1-202777232-G-T). At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |