Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001985744 | SCV002244112 | uncertain significance | Noonan syndrome 8 | 2024-11-27 | criteria provided, single submitter | clinical testing | This sequence change replaces histidine, which is basic and polar, with arginine, which is basic and polar, at codon 109 of the RIT1 protein (p.His109Arg). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with RIT1-related conditions. ClinVar contains an entry for this variant (Variation ID: 1462672). Invitae Evidence Modeling incorporating data from in vitro experimental studies (internal data) indicates that this missense variant is not expected to disrupt RIT1 function with a negative predictive value of 95%. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV002324413 | SCV002610875 | uncertain significance | Cardiovascular phenotype | 2024-09-23 | criteria provided, single submitter | clinical testing | The p.H109R variant (also known as c.326A>G), located in coding exon 4 of the RIT1 gene, results from an A to G substitution at nucleotide position 326. The histidine at codon 109 is replaced by arginine, an amino acid with highly similar properties. This amino acid position is conserved. In addition, this alteration is predicted to be tolerated by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Genome- |
RCV001985744 | SCV004050460 | uncertain significance | Noonan syndrome 8 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
Gene |
RCV004591666 | SCV005081356 | uncertain significance | not provided | 2024-05-30 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis indicates that this missense variant does not alter protein structure/function |