Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001901816 | SCV002173403 | likely benign | Noonan syndrome 9 | 2024-01-19 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002458772 | SCV002616285 | uncertain significance | Cardiovascular phenotype | 2024-08-23 | criteria provided, single submitter | clinical testing | The p.P1183S variant (also known as c.3547C>T), located in coding exon 23 of the SOS2 gene, results from a C to T substitution at nucleotide position 3547. The proline at codon 1183 is replaced by serine, an amino acid with similar properties. This amino acid position is conserved. In addition, the in silico prediction for this alteration is inconclusive. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Genome- |
RCV001901816 | SCV002762989 | uncertain significance | Noonan syndrome 9 | criteria provided, single submitter | clinical testing | ||
ARUP Laboratories, |
RCV001901816 | SCV004563713 | uncertain significance | Noonan syndrome 9 | 2023-08-08 | criteria provided, single submitter | clinical testing | The SOS2 c.3547C>T; p.Pro1183Ser variant (rs139401491), to our knowledge, is not reported in the medical literature but is reported in ClinVar (Variation ID: 1404777). This variant is found in the non-Finnish European population with an allele frequency of 0.0028% (3/106,010 alleles) in the Genome Aggregation Database. Computational analyses are uncertain whether this variant is neutral or deleterious (REVEL: 0.386). Due to limited information, the clinical significance of this variant is uncertain at this time. |