Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV002508208 | SCV002817477 | uncertain significance | not provided | 2022-06-23 | criteria provided, single submitter | clinical testing | Reported in an individual and their father with peripheral neuropathy and Charcot-Marie-Tooth disease type 2 respectively (Auer-Grumbach et al., 2016); Published functional studies suggest p.(A422D) results in decreased protein activity (Auer-Grumbach et al., 2016); In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 34480178, 33144514, 27588448) |
Labcorp Genetics |
RCV002508208 | SCV003525676 | uncertain significance | not provided | 2022-09-06 | criteria provided, single submitter | clinical testing | This sequence change replaces alanine, which is neutral and non-polar, with aspartic acid, which is acidic and polar, at codon 422 of the MME protein (p.Ala422Asp). This variant is present in population databases (rs777476150, gnomAD 0.004%). This missense change has been observed in individual(s) with clinical features of Charcot-Marie-Tooth disease (PMID: 27588448; Invitae). It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 265744). Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be disruptive. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
OMIM | RCV000255510 | SCV000322712 | risk factor | Charcot-Marie-Tooth disease axonal type 2T | 2016-10-06 | no assertion criteria provided | literature only |