Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Quest Diagnostics Nichols Institute San Juan Capistrano | RCV000239340 | SCV000296468 | likely pathogenic | Breast-ovarian cancer, familial, susceptibility to, 1 | 2016-05-18 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001389864 | SCV001591385 | pathogenic | Hereditary breast ovarian cancer syndrome | 2020-01-20 | criteria provided, single submitter | clinical testing | Experimental studies have shown that a different variant at this donor site disrupts mRNA splicing (PMID: 21394826, 18489799). Disruption of this splice site has been observed in individuals with breast and/or ovarian cancer (PMID: 12491499, 21324516, 23767878, 24884479, Invitae). ClinVar contains an entry for this variant (Variation ID: 252396). This variant is not present in population databases (ExAC no frequency). This sequence change affects a donor splice site in intron 14 of the BRCA1 gene. It is expected to disrupt RNA splicing and likely results in an absent or disrupted protein product. For these reasons, this variant has been classified as Pathogenic. Donor and acceptor splice site variants typically lead to a loss of protein function (PMID: 16199547), and loss-of-function variants in BRCA1 are known to be pathogenic (PMID: 20104584). |
Gene |
RCV003327390 | SCV004034878 | pathogenic | not provided | 2023-03-07 | criteria provided, single submitter | clinical testing | Canonical splice site variant predicted to result in a null allele in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge; Also known as 4794+2T>G; This variant is associated with the following publications: (PMID: 23767878, 21324516, 21394826, 24884479, 12491499, 16199547, 18489799, 20104584, 26295337, 30464253) |