Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Evidence- |
RCV000660970 | SCV000783209 | pathogenic | Breast-ovarian cancer, familial, susceptibility to, 1 | 2017-12-15 | reviewed by expert panel | curation | Variant allele predicted to encode a truncated non-functional protein. |
Gene |
RCV000236593 | SCV000293300 | likely pathogenic | not provided | 2015-10-22 | criteria provided, single submitter | clinical testing | This duplication of 4 nucleotides in BRCA1 is denoted c.5054_5057dupCTCA at the cDNA level and p.Val1687SerfsX9 (V1687SfsX9) at the protein level. The normal sequence, with the bases that are duplicated in braces, is ACTA[CTCA]TGTT. The duplication causes a frameshift, which changes a Valine to a Serine at codon 1687, and creates a premature stop codon at position 9 of the new reading frame. Although this variant has not, to our knowledge, been reported in the literature, it is predicted to cause loss of normal protein function through either protein truncation or nonsense-mediated mRNA decay. Based on the currently available information, we consider this duplication to be a likely pathogenic variant. |
Baylor Genetics | RCV000660970 | SCV004215168 | likely pathogenic | Breast-ovarian cancer, familial, susceptibility to, 1 | 2024-02-10 | criteria provided, single submitter | clinical testing |