Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001907678 | SCV002128185 | uncertain significance | Immunodeficiency 31B; Autoimmune enteropathy and endocrinopathy - susceptibility to chronic infections syndrome; Mendelian susceptibility to mycobacterial diseases due to partial STAT1 deficiency | 2022-12-29 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be tolerated. ClinVar contains an entry for this variant (Variation ID: 1364769). This variant has not been reported in the literature in individuals affected with STAT1-related conditions. This variant is present in population databases (rs771750276, gnomAD 0.07%). This sequence change replaces threonine, which is neutral and polar, with serine, which is neutral and polar, at codon 224 of the STAT1 protein (p.Thr224Ser). |
Ambry Genetics | RCV002550325 | SCV003548873 | uncertain significance | Inborn genetic diseases | 2021-10-12 | criteria provided, single submitter | clinical testing | The c.670A>T (p.T224S) alteration is located in exon 9 (coding exon 7) of the STAT1 gene. This alteration results from a A to T substitution at nucleotide position 670, causing the threonine (T) at amino acid position 224 to be replaced by a serine (S). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |