Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001071186 | SCV001236474 | uncertain significance | Bardet-Biedl syndrome | 2022-03-18 | criteria provided, single submitter | clinical testing | This sequence change replaces leucine, which is neutral and non-polar, with proline, which is neutral and non-polar, at codon 11 of the TRIM32 protein (p.Leu11Pro). This variant is present in population databases (rs767818381, gnomAD 0.03%). This variant has not been reported in the literature in individuals affected with TRIM32-related conditions. ClinVar contains an entry for this variant (Variation ID: 864083). Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Fulgent Genetics, |
RCV002482140 | SCV002796887 | uncertain significance | Sarcotubular myopathy; Bardet-Biedl syndrome 11 | 2022-05-22 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV003413896 | SCV004108596 | uncertain significance | TRIM32-related disorder | 2022-11-17 | criteria provided, single submitter | clinical testing | The TRIM32 c.32T>C variant is predicted to result in the amino acid substitution p.Leu11Pro. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.037% of alleles in individuals of Latino descent in gnomAD (http://gnomad.broadinstitute.org/variant/9-119460053-T-C). At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |
Ambry Genetics | RCV004030779 | SCV004972047 | uncertain significance | Inborn genetic diseases | 2024-01-23 | criteria provided, single submitter | clinical testing | The c.32T>C (p.L11P) alteration is located in exon 2 (coding exon 1) of the TRIM32 gene. This alteration results from a T to C substitution at nucleotide position 32, causing the leucine (L) at amino acid position 11 to be replaced by a proline (P). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |