Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000760886 | SCV000890782 | uncertain significance | not provided | 2023-07-28 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene or region of a gene for which loss of function is not a well-established mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |
UNC Molecular Genetics Laboratory, |
RCV001374606 | SCV001571451 | uncertain significance | Autism spectrum disorder | 2020-05-01 | criteria provided, single submitter | research | The SHANK2 c.4906C>T (p.Arg1636*) is a nonsense variant that is predicted to result in early protein truncation. However, this variant is located 74 nucleotides from the intron/exon junction of the penultimate exon of the SHANK2 gene and therefore may escape, or be subject to reduced nonsense-mediated RNA decay (NMD). This variant has not been reported previously in the medical literature or in human population variant databases such as gnomAD, and is considered a variant of uncertain clinical significance. |
Biochemical Molecular Genetic Laboratory, |
RCV001028010 | SCV001190774 | likely pathogenic | Autism, susceptibility to, 17 | 2020-02-05 | no assertion criteria provided | clinical testing |