Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000861443 | SCV001001753 | likely benign | Arrhythmogenic right ventricular dysplasia 13 | 2025-01-28 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001759644 | SCV002005483 | likely benign | not provided | 2020-11-17 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004029297 | SCV003911594 | uncertain significance | not specified | 2023-01-05 | criteria provided, single submitter | clinical testing | The p.R646C variant (also known as c.1936C>T), located in coding exon 13 of the CTNNA3 gene, results from a C to T substitution at nucleotide position 1936. The arginine at codon 646 is replaced by cysteine, an amino acid with highly dissimilar properties. This amino acid position is conserved. In addition, this alteration is predicted to be tolerated by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Prevention |
RCV003948056 | SCV004763457 | likely benign | CTNNA3-related disorder | 2022-09-14 | no assertion criteria provided | clinical testing | This variant is classified as likely benign based on ACMG/AMP sequence variant interpretation guidelines (Richards et al. 2015 PMID: 25741868, with internal and published modifications). |