Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001788438 | SCV002031211 | uncertain significance | not provided | 2021-05-19 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; In-silico analysis, which includes splice predictors and evolutionary conservation, is inconclusive as to whether the variant alters gene splicing. In the absence of RNA/functional studies, the actual effect of this sequence change is unknown.; This variant is associated with the following publications: (PMID: 30773277) |
Labcorp Genetics |
RCV001788438 | SCV003483989 | uncertain significance | not provided | 2022-06-25 | criteria provided, single submitter | clinical testing | This sequence change replaces threonine, which is neutral and polar, with methionine, which is neutral and non-polar, at codon 653 of the TONSL protein (p.Thr653Met). This variant is present in population databases (rs755055463, gnomAD 0.007%). This missense change has been observed in individual(s) with spondylometaphyseal dysplasia (PMID: 30773277). ClinVar contains an entry for this variant (Variation ID: 982207). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Deleterious"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0"). In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
University of Washington Center for Mendelian Genomics, |
RCV001261769 | SCV001439085 | likely pathogenic | Skeletal dysplaisia with extra-skeletal manifestations | no assertion criteria provided | research |