Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
NIHR Bioresource Rare Diseases, |
RCV001027619 | SCV001190191 | likely pathogenic | Inherited Immunodeficiency Diseases | 2019-01-01 | criteria provided, single submitter | research | |
Labcorp Genetics |
RCV001862410 | SCV002297886 | likely pathogenic | Schimke immuno-osseous dysplasia | 2023-11-20 | criteria provided, single submitter | clinical testing | This variant results in the deletion of part of exon 6 (c.1146_1147+2del) of the SMARCAL1 gene. It is expected to disrupt RNA splicing. Variants that disrupt the donor or acceptor splice site typically lead to a loss of protein function (PMID: 16199547), and loss-of-function variants in SMARCAL1 are known to be pathogenic (PMID: 11799392, 20301550). This variant is not present in population databases (gnomAD no frequency). This variant has been observed in individual(s) with Schimke immunoosseous dysplasia (PMID: 15523612, 32499645). ClinVar contains an entry for this variant (Variation ID: 827736). In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. |
Fulgent Genetics, |
RCV001862410 | SCV002815737 | pathogenic | Schimke immuno-osseous dysplasia | 2021-09-17 | criteria provided, single submitter | clinical testing | |
OMIM | RCV001862410 | SCV000024566 | pathogenic | Schimke immuno-osseous dysplasia | 2004-12-01 | no assertion criteria provided | literature only |