Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002631431 | SCV002964113 | pathogenic | not provided | 2023-12-11 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gln1172*) in the TTC37 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in TTC37 are known to be pathogenic (PMID: 20176027, 21120949). This variant is present in population databases (rs780738565, gnomAD 0.009%). This variant has not been reported in the literature in individuals affected with TTC37-related conditions. ClinVar contains an entry for this variant (Variation ID: 1931175). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV002631431 | SCV004167884 | uncertain significance | not provided | 2023-04-12 | criteria provided, single submitter | clinical testing | Has not been previously reported in peer-reviewed literature as pathogenic or benign to our knowledge. However, in an abstract by Polo et al. (2019), p.(Q1172*) was seen phase unknown with a second TTC37 variant in a proband with clinical features of trichohepatoenteric syndrome; Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; This variant is associated with the following publications: (PMID: Polo_2019_Abstract) |