Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
ARUP Laboratories, |
RCV000507571 | SCV000604069 | pathogenic | not specified | 2017-04-08 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002524917 | SCV003484276 | pathogenic | not provided | 2022-11-24 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Experimental studies have shown that this premature translational stop signal affects KIAA0753 function (PMID: 34523780). Algorithms developed to predict the effect of variants on protein structure and function are not available or were not evaluated for this variant. ClinVar contains an entry for this variant (Variation ID: 439846). This premature translational stop signal has been observed in individuals with KIAA0753-related conditions (PMID: 34016807, 34523780). This variant is present in population databases (rs770256450, gnomAD 0.005%). This sequence change creates a premature translational stop signal (p.Arg524Thrfs*7) in the KIAA0753 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in KIAA0753 are known to be pathogenic (PMID: 29138412). |
OMIM | RCV001559338 | SCV001781547 | pathogenic | Short-rib thoracic dysplasia 21 without polydactyly | 2021-10-07 | no assertion criteria provided | literature only |