Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Molecular Genetics Laboratory, |
RCV001172962 | SCV001336037 | uncertain significance | Charcot-Marie-Tooth disease | criteria provided, single submitter | clinical testing | ||
Ambry Genetics | RCV002418604 | SCV002721832 | uncertain significance | Inborn genetic diseases | 2020-08-24 | criteria provided, single submitter | clinical testing | The p.G68S variant (also known as c.202G>A), located in coding exon 3 of the FIG4 gene, results from a G to A substitution at nucleotide position 202. The glycine at codon 68 is replaced by serine, an amino acid with similar properties. This amino acid position is highly conserved in available vertebrate species. In addition, this alteration is predicted to be tolerated by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Labcorp Genetics |
RCV002558741 | SCV003258313 | uncertain significance | Charcot-Marie-Tooth disease type 4 | 2024-09-29 | criteria provided, single submitter | clinical testing | This sequence change replaces glycine, which is neutral and non-polar, with serine, which is neutral and polar, at codon 68 of the FIG4 protein (p.Gly68Ser). This variant is present in population databases (rs745860370, gnomAD 0.002%). This variant has not been reported in the literature in individuals affected with FIG4-related conditions. ClinVar contains an entry for this variant (Variation ID: 916876). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Prevention |
RCV004743317 | SCV005344971 | uncertain significance | FIG4-related disorder | 2024-08-23 | no assertion criteria provided | clinical testing | The FIG4 c.202G>A variant is predicted to result in the amino acid substitution p.Gly68Ser. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0018% of alleles in individuals of European (Non-Finnish) descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |