Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000699331 | SCV000828037 | likely benign | Amyotrophic lateral sclerosis type 4; Spinocerebellar ataxia, autosomal recessive, with axonal neuropathy 2 | 2023-06-21 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004669088 | SCV005162241 | uncertain significance | Inborn genetic diseases | 2024-05-06 | criteria provided, single submitter | clinical testing | The c.3823C>T (p.R1275C) alteration is located in exon 10 (coding exon 8) of the SETX gene. This alteration results from a C to T substitution at nucleotide position 3823, causing the arginine (R) at amino acid position 1275 to be replaced by a cysteine (C). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Prevention |
RCV004733006 | SCV005358242 | uncertain significance | SETX-related disorder | 2024-07-26 | no assertion criteria provided | clinical testing | The SETX c.3823C>T variant is predicted to result in the amino acid substitution p.Arg1275Cys. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.014% of alleles in individuals of Latino descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |