Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001764938 | SCV001989472 | uncertain significance | not provided | 2020-02-10 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; In silico analysis supports that this missense variant does not alter protein structure/function |
Ambry Genetics | RCV002359231 | SCV002648327 | uncertain significance | Inborn genetic diseases | 2020-11-04 | criteria provided, single submitter | clinical testing | The p.A1945V variant (also known as c.5834C>T), located in coding exon 12 of the SETX gene, results from a C to T substitution at nucleotide position 5834. The alanine at codon 1945 is replaced by valine, an amino acid with similar properties. A different alteration at this position (p.A1945P) was reported along with a frameshift mutation in a patient with early onset progressive ataxia (Fogel BL et al. Neurology, 2006 Dec;67:2083-4). This amino acid position is highly conserved in available vertebrate species. In addition, the in silico prediction for this alteration is inconclusive. Based on the supporting evidence, this variant is unlikely to be causative of juvenile amyotrophic lateral sclerosis; however, its contribution to the development of spinocerebellar ataxia with axonal neuropathy is uncertain. |
Genome- |
RCV003234099 | SCV003931294 | uncertain significance | Spinocerebellar ataxia, autosomal recessive, with axonal neuropathy 2 | 2023-02-08 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV003234100 | SCV003931295 | uncertain significance | Amyotrophic lateral sclerosis type 4 | 2023-02-08 | criteria provided, single submitter | clinical testing |