Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001314393 | SCV001504926 | uncertain significance | Familial aplasia of the vermis; Meckel-Gruber syndrome | 2021-09-01 | criteria provided, single submitter | clinical testing | This sequence change replaces proline with leucine at codon 1182 of the RPGRIP1L protein (p.Pro1182Leu). The proline residue is moderately conserved and there is a moderate physicochemical difference between proline and leucine. This variant is not present in population databases (ExAC no frequency). This variant has not been reported in the literature in individuals affected with RPGRIP1L-related conditions. Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV004034311 | SCV005015374 | uncertain significance | Inborn genetic diseases | 2023-10-16 | criteria provided, single submitter | clinical testing | The c.3545C>T (p.P1182L) alteration is located in exon 24 (coding exon 23) of the RPGRIP1L gene. This alteration results from a C to T substitution at nucleotide position 3545, causing the proline (P) at amino acid position 1182 to be replaced by a leucine (L). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Natera, |
RCV001836291 | SCV002087504 | uncertain significance | Familial aplasia of the vermis | 2020-02-04 | no assertion criteria provided | clinical testing | |
Prevention |
RCV004733247 | SCV005365975 | uncertain significance | RPGRIP1L-related disorder | 2024-04-17 | no assertion criteria provided | clinical testing | The RPGRIP1L c.3545C>T variant is predicted to result in the amino acid substitution p.Pro1182Leu. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0033% of alleles in individuals of South Asian descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |