Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001008662 | SCV001168441 | pathogenic | not provided | 2019-01-22 | criteria provided, single submitter | clinical testing | The W1359X nonsense variant in the MED13L gene has been reported previously as a de novo change in association with autism spectrum disorder (Wang et al., 2016; Isossifov et al., 2014). This pathogenic variant is predicted to cause loss of normal protein function either through protein truncation or nonsense-mediated mRNA decay. This variant is not observed in large population cohorts (Lek., 2016). Therefore the W1359X is a pathogenic variant. |
Genome |
RCV001265126 | SCV001443159 | pathogenic | Cardiac anomalies - developmental delay - facial dysmorphism syndrome | 2016-06-21 | no assertion criteria provided | provider interpretation | Submission from Simons Searchlight facilitated by GenomeConnect. Variant interpreted by the Simons Searchlight team most recently on 2016-06-21 and interpreted as Pathogenic. Variant was initially reported by the University of Washington TIGER Study and was later confirmed by GeneDx. CK3+CK19626957The reporting laboratory might also submit to ClinVar. |
University of Washington Center for Mendelian Genomics, |
RCV001291369 | SCV001479843 | likely pathogenic | Autism spectrum disorder | no assertion criteria provided | research |