Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000005039 | SCV001201672 | pathogenic | Hypercholesterolemia, familial, 4 | 2023-09-27 | criteria provided, single submitter | clinical testing | This premature translational stop signal has been observed in individual(s) with autosomal recessive hypercholesterolemia (PMID: 11326085). It has also been observed to segregate with disease in related individuals. This sequence change creates a premature translational stop signal (p.Trp22*) in the LDLRAP1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in LDLRAP1 are known to be pathogenic (PMID: 11326085, 12464675). This variant is not present in population databases (gnomAD no frequency). ClinVar contains an entry for this variant (Variation ID: 4773). For these reasons, this variant has been classified as Pathogenic. |
Revvity Omics, |
RCV000005039 | SCV002017137 | pathogenic | Hypercholesterolemia, familial, 4 | 2024-01-03 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV000005039 | SCV002776831 | pathogenic | Hypercholesterolemia, familial, 4 | 2021-09-08 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV000005039 | SCV004173334 | pathogenic | Hypercholesterolemia, familial, 4 | 2023-04-11 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000005039 | SCV000025215 | pathogenic | Hypercholesterolemia, familial, 4 | 2001-05-18 | no assertion criteria provided | literature only |