Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
UW Hindbrain Malformation Research Program, |
RCV000201547 | SCV000256480 | pathogenic | Joubert syndrome 18 | 2015-02-23 | criteria provided, single submitter | research | |
Labcorp Genetics |
RCV000796275 | SCV000935781 | pathogenic | Orofacial-digital syndrome IV; Joubert syndrome 18 | 2024-10-02 | criteria provided, single submitter | clinical testing | This sequence change affects the initiator methionine of the TCTN3 mRNA. The next in-frame methionine is located at codon 152. This variant is present in population databases (rs745688122, gnomAD 0.02%). Disruption of the initiator codon has been observed in individual(s) with personal or family history of Joubert syndrome (PMID: 26092869; internal data). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 217703). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV003227713 | SCV003924545 | likely pathogenic | not provided | 2022-11-10 | criteria provided, single submitter | clinical testing | Also reported with a variant on the opposite allele (in trans) in a patient with features of a TCTN3-related disorder in the published literature (Huljev Frkovic et al., 2022); Initiation codon variant in a gene for which loss of function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 26092869, 35170189) |
Eurofins- |
RCV000201547 | SCV003935115 | likely pathogenic | Joubert syndrome 18 | 2022-12-13 | criteria provided, single submitter | clinical testing | |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV004782310 | SCV005395372 | pathogenic | Joubert syndrome and related disorders | 2024-09-19 | criteria provided, single submitter | clinical testing | Variant summary: TCTN3 c.3G>A (p.Met1Ile) alters the initiation codon and is predicted to result either in absence of the protein or truncation of the encoded protein due to translation initiation at a downstream codon. The variant allele was found at a frequency of 3.2e-05 in 155526 control chromosomes. c.3G>A has been reported in the literature in individuals affected with Joubert Syndrome And Related Disorders. These data indicate that the variant is likely to be associated with disease. To our knowledge, no experimental evidence demonstrating an impact on protein function has been reported. The following publications have been ascertained in the context of this evaluation (PMID: 26092869, 35170189, 37217489). ClinVar contains an entry for this variant (Variation ID: 217703). Based on the evidence outlined above, the variant was classified as pathogenic. |
Fulgent Genetics, |
RCV000796275 | SCV005678478 | likely pathogenic | Orofacial-digital syndrome IV; Joubert syndrome 18 | 2024-01-23 | criteria provided, single submitter | clinical testing |