Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001236792 | SCV001409528 | likely benign | Lethal congenital glycogen storage disease of heart | 2024-10-20 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002451571 | SCV002617593 | uncertain significance | Cardiovascular phenotype | 2021-09-26 | criteria provided, single submitter | clinical testing | The p.R116H variant (also known as c.347G>A), located in coding exon 3 of the PRKAG2 gene, results from a G to A substitution at nucleotide position 347. The arginine at codon 116 is replaced by histidine, an amino acid with highly similar properties. This amino acid position is conserved. In addition, the in silico prediction for this alteration is inconclusive. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |
Gene |
RCV005054348 | SCV005688059 | uncertain significance | not provided | 2024-07-31 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis indicates that this missense variant does not alter protein structure/function |