Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001429078 | SCV001631789 | likely benign | Adams-Oliver syndrome 5 | 2024-09-04 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001569885 | SCV001794051 | uncertain significance | not provided | 2024-08-18 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Identified in a patient with ischemic stroke in published literature (PMID: 36973604); This variant is associated with the following publications: (PMID: 36973604) |
ARUP Laboratories, |
RCV001569885 | SCV004564874 | likely benign | not provided | 2023-11-10 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004038272 | SCV005027207 | uncertain significance | Familial thoracic aortic aneurysm and aortic dissection | 2024-02-06 | criteria provided, single submitter | clinical testing | The p.G1088S variant (also known as c.3262G>A), located in coding exon 20 of the NOTCH1 gene, results from a G to A substitution at nucleotide position 3262. The glycine at codon 1088 is replaced by serine, an amino acid with similar properties. This amino acid position is well conserved in available vertebrate species. In addition, the in silico prediction for this alteration is inconclusive. Based on the available evidence, the clinical significance of this alteration remains unclear. |