Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Revvity Omics, |
RCV001781254 | SCV002018344 | pathogenic | not provided | 2019-09-13 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001851821 | SCV002247240 | pathogenic | Adams-Oliver syndrome 5 | 2021-09-16 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 12476). This variant is also known as R1108X. This premature translational stop signal has been observed in individual(s) with NOTCH1-related conditions (PMID: 16025100, 23798201, 31654484). It has also been observed to segregate with disease in related individuals. This variant is not present in population databases (ExAC no frequency). This sequence change creates a premature translational stop signal (p.Arg1107*) in the NOTCH1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in NOTCH1 are known to be pathogenic (PMID: 16025100, 21457232, 25132448, 25963545). |
Gene |
RCV001781254 | SCV005201606 | pathogenic | not provided | 2023-11-15 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 23798201, 34065301, 31654484, 35947102, 16025100) |
OMIM | RCV000013294 | SCV000033541 | pathogenic | Aortic valve disease 1 | 2005-09-08 | no assertion criteria provided | literature only |