Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000428861 | SCV000536587 | uncertain significance | not provided | 2024-03-27 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant does not alter protein structure/function; Identified in a patient with a connective tissue disorder who harbored a second variant in the DCHS1 gene (PMID: 30675029); This variant is associated with the following publications: (PMID: 30675029, 37839360, 38100419) |
Labcorp Genetics |
RCV000471204 | SCV000548919 | likely benign | Adams-Oliver syndrome 5 | 2025-01-26 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002339103 | SCV002640627 | uncertain significance | Familial thoracic aortic aneurysm and aortic dissection | 2024-12-29 | criteria provided, single submitter | clinical testing | The p.A1742S variant (also known as c.5224G>T), located in coding exon 28 of the NOTCH1 gene, results from a G to T substitution at nucleotide position 5224. The alanine at codon 1742 is replaced by serine, an amino acid with similar properties. This variant was reported in an individual in a hereditary aortopathy cohort, but the clinical details were limited (Renner S et al. Genet Med, 2019 Aug;21:1832-1841). This amino acid position is not well conserved in available vertebrate species. In addition, this alteration is predicted to be tolerated by in silico analysis. Based on the available evidence, the clinical significance of this variant remains unclear. |