Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Center for Advanced Laboratory Medicine, |
RCV000852763 | SCV000995481 | likely benign | Ventricular tachycardia | 2019-01-31 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001858513 | SCV002168600 | uncertain significance | Progressive familial heart block type IB | 2024-09-16 | criteria provided, single submitter | clinical testing | This sequence change replaces arginine, which is basic and polar, with histidine, which is basic and polar, at codon 755 of the TRPM4 protein (p.Arg755His). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the gnomAD database. This variant has not been reported in the literature in individuals affected with TRPM4-related conditions. ClinVar contains an entry for this variant (Variation ID: 691739). An algorithm developed to predict the effect of missense changes on protein structure and function outputs the following: PolyPhen-2: "Benign". The histidine amino acid residue is found in multiple mammalian species, which suggests that this missense change does not adversely affect protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV002442789 | SCV002733416 | uncertain significance | Cardiovascular phenotype | 2023-10-10 | criteria provided, single submitter | clinical testing | The p.R755H variant (also known as c.2264G>A), located in coding exon 17 of the TRPM4 gene, results from a G to A substitution at nucleotide position 2264. The arginine at codon 755 is replaced by histidine, an amino acid with highly similar properties. This amino acid position is poorly conserved in available vertebrate species. In addition, this alteration is predicted to be tolerated by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this alteration remains unclear. |