Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001519216 | SCV001728046 | benign | not provided | 2025-02-03 | criteria provided, single submitter | clinical testing | |
Breakthrough Genomics, |
RCV001519216 | SCV005268834 | benign | not provided | criteria provided, single submitter | not provided | ||
ARUP Laboratories, |
RCV001519216 | SCV005878385 | benign | not provided | 2024-07-30 | criteria provided, single submitter | clinical testing | |
Department of Pathology and Laboratory Medicine, |
RCV001356400 | SCV001551559 | benign | not specified | no assertion criteria provided | clinical testing | The SAMD9 p.R75W variant was not identified in the literature nor was it identified in ClinVar. The variant was identified in dbSNP (ID: rs111780648) and COSMIC (tissue: stomach). The variant was identified in control databases in 5540 of 282718 chromosomes (87 homozygous) at a frequency of 0.01960 (Genome Aggregation Database March 6, 2019, v2.1.1). The p.R75 residue is not highly conserved in mammals and computational analyses (MUT Assesor, PolyPhen-2, SIFT, MutationTaster, Revel, FATHMM, MetaLR, DANN) do not suggest a high likelihood of impact to the protein. The variant occurs outside of the splicing consensus sequence and in silico or computational prediction software programs (Splice AI exome) do not predict a deleterious effect on splicing. In summary, based on the above information this variant meets our laboratory's criteria to be classified as benign. |