Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000729482 | SCV000857149 | uncertain significance | not provided | 2017-09-28 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV001578718 | SCV001806007 | uncertain significance | Bardet-Biedl syndrome 13 | 2021-07-14 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV001578755 | SCV001806056 | uncertain significance | Joubert syndrome 28 | 2021-07-14 | criteria provided, single submitter | clinical testing | |
Genome- |
RCV001578756 | SCV001806057 | uncertain significance | Meckel syndrome, type 1 | 2021-07-14 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002535120 | SCV003270198 | uncertain significance | Familial aplasia of the vermis; Meckel-Gruber syndrome | 2022-08-05 | criteria provided, single submitter | clinical testing | This sequence change replaces threonine, which is neutral and polar, with methionine, which is neutral and non-polar, at codon 243 of the MKS1 protein (p.Thr243Met). This variant is present in population databases (rs749668169, gnomAD 0.006%). This variant has not been reported in the literature in individuals affected with MKS1-related conditions. ClinVar contains an entry for this variant (Variation ID: 594241). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is expected to disrupt MKS1 protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Prevention |
RCV004733027 | SCV005360965 | uncertain significance | MKS1-related disorder | 2024-08-19 | no assertion criteria provided | clinical testing | The MKS1 c.728C>T variant is predicted to result in the amino acid substitution p.Thr243Met. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0065% of alleles in individuals of South Asian descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |