Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002513634 | SCV003440803 | pathogenic | Charcot-Marie-Tooth disease type 4A | 2022-11-14 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Experimental studies have shown that this missense change affects GDAP1 function (PMID: 21753178, 32183277). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Tolerated"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0"). ClinVar contains an entry for this variant (Variation ID: 50557). This missense change has been observed in individuals with autosomal dominant Charcot-Marie-Tooth disease (PMID: 21753178, 28244113). It has also been observed to segregate with disease in related individuals. This variant is not present in population databases (gnomAD no frequency). This sequence change replaces alanine, which is neutral and non-polar, with glycine, which is neutral and non-polar, at codon 156 of the GDAP1 protein (p.Ala156Gly). |
OMIM | RCV000043548 | SCV000071289 | pathogenic | Charcot-Marie-Tooth disease axonal type 2K | 2011-08-09 | no assertion criteria provided | literature only | |
Inherited Neuropathy Consortium Ii, |
RCV000043548 | SCV004174644 | uncertain significance | Charcot-Marie-Tooth disease axonal type 2K | 2016-01-06 | no assertion criteria provided | literature only |