Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV004385867 | SCV004867218 | uncertain significance | Inborn genetic diseases | 2023-11-18 | criteria provided, single submitter | clinical testing | The c.1124G>A (p.R375Q) alteration is located in exon 9 (coding exon 9) of the F7 gene. This alteration results from a G to A substitution at nucleotide position 1124, causing the arginine (R) at amino acid position 375 to be replaced by a glutamine (Q). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Neuberg Centre For Genomic Medicine, |
RCV004771549 | SCV005382442 | uncertain significance | Congenital factor VII deficiency | 2023-05-20 | criteria provided, single submitter | clinical testing | The missense variant c.1058G>A(p.Arg353Gln) in F7 gene has been reported as a polymorphic marker in individual(s) with F7 related disorders (Pushkov et al. 2011; Lane A et. al., 1996). The observed variant has allele frequency of 0.005% in gnomAD exomes database. This variant has not been submitted to the ClinVar database. Multiple lines of computational evidence (Polyphen - Benign, SIFT - Tolerated and MutationTaster - Polymorphism) predict no damaging effect on protein structure and function for this variant. The amino acid Arg at position 353 is changed to a Gln changing protein sequence and it might alter its composition and physico-chemical properties. For these reasons, this variant has been classified as Uncertain Significance (VUS). In the absence of another reportable variant in F7 gene, the molecular diagnosis is not confirmed. |