Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV002510792 | SCV002820531 | likely pathogenic | not provided | 2023-01-10 | criteria provided, single submitter | clinical testing | Identified with a second TUBGCP6 variant in a patient with microcephaly with simplified gyral pattern, retinal dystrophy, congenital heart defects, and triphalangeal thumbs in published literature (Martin et al., 2014); Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 25344692, 34426522, Chen2022[CaseReport]) |
Labcorp Genetics |
RCV002510792 | SCV003444609 | pathogenic | not provided | 2024-05-28 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg739*) in the TUBGCP6 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in TUBGCP6 are known to be pathogenic (PMID: 25344692). This variant is present in population databases (rs724159975, gnomAD 0.007%). This premature translational stop signal has been observed in individual(s) with microcephalic primordial dwarfism and centriole dysfunction (PMID: 25344692). ClinVar contains an entry for this variant (Variation ID: 162403). For these reasons, this variant has been classified as Pathogenic. |
OMIM | RCV000149798 | SCV000196607 | pathogenic | Microcephaly and chorioretinopathy 1 | 2014-12-01 | no assertion criteria provided | literature only |