Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV004383751 | SCV004869902 | uncertain significance | Inborn genetic diseases | 2024-01-17 | criteria provided, single submitter | clinical testing | The c.2161G>C (p.A721P) alteration is located in exon 13 (coding exon 13) of the FANCM gene. This alteration results from a G to C substitution at nucleotide position 2161, causing the alanine (A) at amino acid position 721 to be replaced by a proline (P). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Labcorp Genetics |
RCV005104412 | SCV005734877 | uncertain significance | Fanconi anemia | 2025-01-03 | criteria provided, single submitter | clinical testing | This sequence change replaces alanine, which is neutral and non-polar, with proline, which is neutral and non-polar, at codon 721 of the FANCM protein (p.Ala721Pro). This variant is present in population databases (rs563314173, gnomAD 0.0009%). This variant has not been reported in the literature in individuals affected with FANCM-related conditions. ClinVar contains an entry for this variant (Variation ID: 3092834). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |