Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001380939 | SCV001579165 | pathogenic | Fanconi anemia | 2022-08-08 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 1069183). This variant has not been reported in the literature in individuals affected with FANCM-related conditions. This variant is present in population databases (rs751954386, gnomAD 0.007%). This sequence change creates a premature translational stop signal (p.Arg1424*) in the FANCM gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in FANCM are known to be pathogenic (PMID: 29895858, 30075111). |
Gene |
RCV001564781 | SCV001787995 | likely pathogenic | not provided | 2023-05-18 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); Has not been previously published as pathogenic or benign to our knowledge |
Quest Diagnostics Nichols Institute San Juan Capistrano | RCV001564781 | SCV005626308 | pathogenic | not provided | 2024-09-07 | criteria provided, single submitter | clinical testing | The FANCM c.4270C>T (p.Arg1424*) variant causes the premature termination of FANCM protein synthesis. This variant has been reported in the published literature in individuals with breast cancer (PMID: 37444426 (2023), 33471991 (2021), see also LOVD (http://databases.lovd.nl/shared)). This variant has also been observed in a reportedly healthy individual (PMID: 33471991 (2021), see also LOVD (http://databases.lovd.nl/shared)). The frequency of this variant in the general population, 0.000035 (4/113276 chromosomes (Genome Aggregation Database, http://gnomad.broadinstitute.org)), is uninformative in the assessment of its pathogenicity. Based on the available information, this variant is classified as pathogenic. |