Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Revvity Omics, |
RCV003144961 | SCV003830618 | likely pathogenic | Vici syndrome | 2022-10-26 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV003395713 | SCV004120806 | likely pathogenic | EPG5-related disorder | 2022-08-26 | criteria provided, single submitter | clinical testing | The EPG5 c.5304+1G>A variant is predicted to disrupt the GT donor site and interfere with normal splicing. To our knowledge, this variant has not been reported in the literature or in a large population database (http://gnomad.broadinstitute.org), indicating this variant is rare. Variants that disrupt the consensus splice donor site in EPG5 are expected to be pathogenic. This variant is interpreted as likely pathogenic. |