Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000556590 | SCV000650813 | uncertain significance | Fanconi anemia complementation group E | 2022-08-15 | criteria provided, single submitter | clinical testing | This sequence change replaces leucine, which is neutral and non-polar, with methionine, which is neutral and non-polar, at codon 531 of the FANCE protein (p.Leu531Met). This variant is present in population databases (rs147390386, gnomAD 0.06%). This variant has not been reported in the literature in individuals affected with FANCE-related conditions. ClinVar contains an entry for this variant (Variation ID: 471923). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Deleterious"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0"). In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Fulgent Genetics, |
RCV000556590 | SCV002816675 | uncertain significance | Fanconi anemia complementation group E | 2021-12-07 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV003424132 | SCV004117273 | uncertain significance | FANCE-related disorder | 2023-09-22 | criteria provided, single submitter | clinical testing | The FANCE c.1591T>A variant is predicted to result in the amino acid substitution p.Leu531Met. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.064% of alleles in individuals of African descent in gnomAD (http://gnomad.broadinstitute.org/variant/6-35434102-T-A). Although we suspect that this variant may be benign, at this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |