Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
3billion, |
RCV002283694 | SCV002572680 | uncertain significance | Autosomal recessive nonsyndromic hearing loss 12 | 2022-09-01 | criteria provided, single submitter | clinical testing | The variant is observed at an extremely low frequency in the gnomAD v2.1.1 dataset (total allele frequency: <0.001%). While this variant results in missense change, protein truncation variants are a common disease-causing mechanism. In silico tool predictions suggest damaging effect of the variant on gene or gene product (REVEL: 0.89; 3Cnet: 0.77). Therefore, this variant is classified as uncertain significance according to the recommendation of ACMG/AMP guideline. |
Institute of Rare Diseases, |
RCV002283694 | SCV005687009 | likely pathogenic | Autosomal recessive nonsyndromic hearing loss 12 | 2025-01-09 | criteria provided, single submitter | research | PM3_Strong;PM2_Supporting;PP3 |
Labcorp Genetics |
RCV005096036 | SCV005824372 | uncertain significance | not provided | 2024-05-18 | criteria provided, single submitter | clinical testing | This sequence change replaces glycine, which is neutral and non-polar, with serine, which is neutral and polar, at codon 185 of the CDH23 protein (p.Gly185Ser). This variant is present in population databases (rs375465342, gnomAD 0.007%). This variant has not been reported in the literature in individuals affected with CDH23-related conditions. ClinVar contains an entry for this variant (Variation ID: 1705380). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) has been performed at Invitae for this missense variant, however the output from this modeling did not meet the statistical confidence thresholds required to predict the impact of this variant on CDH23 protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |