Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001233551 | SCV001406152 | pathogenic | not provided | 2019-10-31 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Leu482Trpfs*40) in the ABCG8 gene. It is expected to result in an absent or disrupted protein product. This variant is present in population databases (rs748233241, ExAC 0.003%). This variant has been observed in an individual affected with sitosterolemia (Invitae). Loss-of-function variants in ABCG8 are known to be pathogenic (PMID: 11452359, 15375183, 16029460). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV001233551 | SCV003837080 | pathogenic | not provided | 2022-08-24 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 33204594) |
Genomic Medicine Center of Excellence, |
RCV003989650 | SCV004808135 | pathogenic | Sitosterolemia 1 | 2024-03-29 | criteria provided, single submitter | clinical testing |