Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001048825 | SCV001212849 | pathogenic | Charcot-Marie-Tooth disease type 4 | 2024-10-30 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Gln460*) in the SH3TC2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in SH3TC2 are known to be pathogenic (PMID: 20220177, 27068304). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with Charcot-Marie-Tooth disease (PMID: 25614874). ClinVar contains an entry for this variant (Variation ID: 637510). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. For these reasons, this variant has been classified as Pathogenic. |
Molecular Diagnostics Laboratory, |
RCV001171314 | SCV001334044 | pathogenic | Charcot-Marie-Tooth disease type 4C | 2020-02-26 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV002501035 | SCV002801229 | likely pathogenic | Charcot-Marie-Tooth disease type 4C; Susceptibility to mononeuropathy of the median nerve, mild | 2021-08-24 | criteria provided, single submitter | clinical testing | |
Inherited Neuropathy Consortium | RCV000789719 | SCV000929095 | pathogenic | Charcot-Marie-Tooth disease | no assertion criteria provided | literature only |