Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001298956 | SCV001488027 | uncertain significance | Jeune thoracic dystrophy; Nephronophthisis | 2024-11-16 | criteria provided, single submitter | clinical testing | This sequence change replaces glutamine, which is neutral and polar, with arginine, which is basic and polar, at codon 869 of the TTC21B protein (p.Gln869Arg). This variant is present in population databases (rs137926033, gnomAD 0.02%). This missense change has been observed in individual(s) with clinical features of Meckel-Gruber syndrome (PMID: 21258341). ClinVar contains an entry for this variant (Variation ID: 1002519). Invitae Evidence Modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) indicates that this missense variant is not expected to disrupt TTC21B protein function with a negative predictive value of 95%. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Gene |
RCV001549692 | SCV001769890 | uncertain significance | not provided | 2024-05-07 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant does not alter protein structure/function; Reported in an individual with a Meckel-Gruber-like phenotype in the literature (PMID: 21258341); This variant is associated with the following publications: (PMID: 21258341, 31764884) |
Fulgent Genetics, |
RCV002486149 | SCV002778746 | uncertain significance | Asphyxiating thoracic dystrophy 4; Nephronophthisis 12 | 2024-04-11 | criteria provided, single submitter | clinical testing |