Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000579181 | SCV000681007 | pathogenic | not provided | 2021-03-13 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; Not observed in large population cohorts (Lek et al., 2016); Has not been previously published as pathogenic or benign to our knowledge |
Ce |
RCV000579181 | SCV001248990 | pathogenic | not provided | 2018-01-01 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002529047 | SCV003307785 | pathogenic | Kleefstra syndrome 1 | 2023-08-04 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 489033). This variant has not been reported in the literature in individuals affected with EHMT1-related conditions. This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Gln170*) in the EHMT1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in EHMT1 are known to be pathogenic (PMID: 16826528, 19264732). |
Laboratoire de Génétique Moléculaire, |
RCV002529047 | SCV003836705 | pathogenic | Kleefstra syndrome 1 | 2021-01-18 | criteria provided, single submitter | clinical testing |